Fgf21 maintains glucose homeostasis by mediating the crosstalk between liver and brain during prolonged fasting
- In: SUBMITTED ORALS on Wednesday, 16 April 2014, 14:30-15:30
- At: PSWC, Melbourne (Australia) (2014)
- Type: Presentation
- By: LIANG, Qingning (The University of Hong Kong, Medicine, Hong kong, China - Hong-Kong)
- Co-author(s): Zhong, Ling (The University of Hong Kong, Hong kong, China - Hong-Kong)
Zhang, Jialiang (The University of Hong Kong, Hong kong, China - Hong-Kong)
Wang, Yu (The University of Hong Kong, Hong kong, China - Hong-Kong)
Tse, Hung-Fat (The University of Hong Kong, Hong kong, China - Hong-Kong)
Wu, Donghai (Guangzhou Institute of Biomedicine and Health, Chinese Academy of Sciences, Guangzhou, China - Hong-Kong)
Bornstein, Stefan (Department of Medicine, University of Dresden, Dresden, Germany)
Lam, Karen (The University of Hong Kong, Hong kong, China - Hong-Kong)
Xu, Aimin (The University of Hong Kong, Hong kong, China - Hong-Kong) - Abstract:
I. Background Hepatic gluconeogenesis is a main source of blood glucose during prolonged fasting, and is orchestrated by endocrine and neural pathways. Fibroblast growth factor 21 (FGF21), a hepatocyte-secreted endocrine factor induced by peroxisome proliferator-pctivated Receptor (PPAR) α during prolonged fasting, has recently been shown to be an
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Last update 28 September 2023