Targeting TRPM2 Signaling to Relieve Brain Pericyte Injury
- In: Short Communications A3 on Tuesday, 23 May 2017, 11:15-12:30
- At: Stockholm (Sweden) (2017)
- Type: Presentation
- By: LU, Yingmei (Zhejiang University City College, Hangzhou, China)
- Co-author(s): Lingxiao Shao: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Quan Jiang: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Ninghe Sun: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Danyang Chen: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Xiuxiu Liu: School of Medicine, Zhejiang University City College, HangZhou, China
Yue Pan: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Yijie Liu: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Fangying Yu: College of pharmaceutical sciences, Zhejiang university, HangZhou, China
Feng Han: College of pharmaceutical sciences, Zhejiang university, HangZhou, China - Abstract:
Backgrounds
Dysfunction of neurovascular pericytes underlies the breakdown of the blood-brain barrier, but the molecular mechanisms are largely unknown.
Aims
In this study, we evaluated the role of the TRPM2 (transient receptor potential melastatin-related 2) channel and autophagy during brain pericyte injury in vitro and in vivo.
Methods
Moni
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